СОСТОЯНИЕ ПРОБЛЕМЫ ОЦЕНКИ ВОЛЕМИЧЕСКОГО СТАТУСА У ПАЦИЕНТОВ В ОСТРОМ ПЕРИОДЕ ИШЕМИЧЕСКОГО ИНСУЛЬТА
Обзорная статья
Дата публикации: марта 28, 2024
Ключевые слова
волемический статус
инсульт
гиперволемия
инсульт
гиперволемия
Как цитировать
Михайлов Е. В., Пасечник И. Н., Корочкина Г. В., Рафаэлова О. Г., Курильченко Д. С. СОСТОЯНИЕ ПРОБЛЕМЫ ОЦЕНКИ ВОЛЕМИЧЕСКОГО СТАТУСА У ПАЦИЕНТОВ В ОСТРОМ ПЕРИОДЕ ИШЕМИЧЕСКОГО ИНСУЛЬТА // Кремлевская медицина. Клинический вестник. 2024. Т. № 1. С. 117-126.
Аннотация
Волемический статус является интегральным понятием, используемым в клинической практике для оценки степени адекватности функции различных звеньев органной системы транспорта кислорода уровню тканевой перфузии и потребления этого субстрата. Восстановление кровоснабжения в ишемизированной ткани – первостепенная задача в остром периоде ишемического инсульта. Динамичная и точная оценка волемического статуса может быть использована в качестве основы целенаправленного лечения и прогностического критерия. В настоящее время отсутствует тот самый «золотой стандарт» оценки уровня волемии, который был бы корректным, безопасным, простым в применении и недорогим. Авторами предпринята попытка проанализировать наиболее изученные методы оценки волемического статуса применительно к острому периоду ишемического инсульта.Литература
1. Савин И.А. и др. Водно-электролитные нарушения в нейрореанимации. – Москва, 2015. – С. 332. [Savin I.A. et al. Water-electrolyte disorders in neuro-resuscitation. – Moscow, 2015. – P. 332. In Russian].
2. Cordemans C. et al. Aiming for a negative fluid balance in patients with acute lung injury and increased intra-abdominal pressure: a pilot study looking at the effects of PAL-treatment // Ann. Intensive Care. – 2012. – No. 5. – Suppl. 1. DOI: 10.1186/2110-5820-2-S1-S15.
3. Бобовник С.В. и др. Периоперационная инфузионная терапия у взрослых // Анестезиология и реаниматология. – 2021. – № 4. – С. 17–33. [Bobovnik S.V. et al. Perioperative fluid therapy in adults // Anesteziologiya I Reanimatologiya (Russian Journal of Anaesthesiology and Reanimatology). – 2021. – No. 4. – P. 17–33. In Russain]. DOI: 10.17116/anaesthesiology202104117.
4. Методические рекомендации Министерства здравоохранения РФ. Принципы периоперационной инфузионной терапии у взрослых пациентов. – 2018. – С. 57. [Methodological recommendations of the Ministry of Health of the Russian Federation. Principles of perioperative infusion therapy in adult patients. – 2018. – P. 57. In Russian].
5. Максимович Н.Е. и др. Патофизиология системы крови и гемостаза. – Гродно: УО «ГрГМУ», 2010. – С. 344. [Maksimovich N.E. et al. Pathophysiology of the blood system and hemostasis. Grodno: EI «GSMU», 2010. – P. 344. In Russian].
6. Rosenberg A.L. et al. Review of a large clinical series: association of cumulative fluid balance on outcome in acute lung injury: a retrospective review of the ARDSnet tidal volume study cohort // J. Intensive Care Med. – 2009. – V. 24. – No. 1. – P. 35–46. DOI: 10.1177/0885066608329850.
7. Кузьков В.В. и др. Инвазивный мониторинг гемодинаники. – Архангельск: Северный государственный медицинский университет, 2008. – С. 392. [Kuzkov V.V. et al. Invasive Hemodynamic Monitoring. Arhangelsk: Northern State Medical University, 2008. – P. 392. In Russian].
8. Задворнов А.А. и др. Клиническая патофизиология отека головного мозга (часть 1) // Вестник анестезиологии и реаниматологии. – 2017. – Т. 14. – № 3. – С. 44–50. [Zadvornov A.A. et al. Clinical pathophysiology of celebral edems (part 1) // Messenger of Anesthesiology and Resuscitation. – 2017. – V. 14. – No. 3. – P. 44–50. In Russian]. DOI: 10.21292/2078-5658-2017-14-3-44-50.
9. Ершов В.И. и др. Гиповолемическая гипернатриемия в дебюте тяжелого ишемического инсульта как предиктор неблагоприятного исхода // Клиника нервных и психических заболеваний. – 2016. – № 6. – С. 10–13. [Ershov V.I. et al. Hypovolemic hyponatremia at the onset of severe ischemic stroke as a predictor of adverse outcome // Zhurnal Nevrologii i Psikhiatrii imeni S.S. Korsakova. – 2016. – No. 6. – P. 10–13. In Russian]. DOI: 10.17116/jnevro20161166110-13.
10. Widimsky P. What is new in ischemic stroke? // Dialogues in Cardiovascular Medicine. – 2017. – V. 22. – No. 3. – P. 25–26.
11. Гончар И.А. Биохимические предикторы и маркеры инфаркта головного мозга. – Минск: БелМАПО, 2013. – С. 512. [Gontschar I.A. et al. Biochemical predictors and markers of ischemic stroke. – Minsk: BMAPE, 2013. – P. 512. In Russian].
12. Nag S. et al. Review: molecular pathogenesis of blood-brain barrier breakdown in acute brain injury // Neuropathology and Applied Neurobiology. – 2011. – V. 37. – No. 9. – P. 3–23.
13. Stokum J.A. et al. Molecular pathophysiology of cerebral edema // Journal Blood Flow Metabolism. – 2016. – V. 36. – No. 3. – P. 513–538.
14. Stokum J.A. et al. Mechanisms of astrocyte-mediated cerebral edema // Neurochemical Research. – 2015. – V. 40. – No. 2. – P. 317–328.
15. Thrane A.S. et al. Drowning stars: reassessing the role of astrocytes in brain edema // Trends in Neuroscience. – 2014. – V. 37. – No. 11. – P. 620–628.
16. Кулеш А.А. и др. Связь между интегральной оценкой магнитно-резонансных маркеров церебральной болезни мелких сосудов, клиническим и функциональным статусом в остром периоде ишемического инсульта // Неврология, нейропсихиатрия, психосоматика. – 2018. – Т. 10. – № 1. – С. 24–31. [Kulesh A.A. et al. A relationship between the integrated assessment of magnetic resonance imaging markers for cerebral small vessel disease and the clinical and functional status in the acute period of ischemic stroke // Nevrologiya, neiropsikhiatriya, psikhosomatika (Neurology, Neuropsychiatry, Psychosomatics). – 2018. – V. 10. – No. 1. – P. 24–31. In Russian]. DOI: 0.14412/2074-2711-2018-1-24-31.
17. Филимонова П.А. и др. Внутрибольничный инсульт у пациентов после кардиохирургических операций и инвазивных вмешательств на сердце // Неврология, нейропсихиатрия, психосоматика. – 2017. – Т. 9. – № 4. – С. 38–45. [Filimonova P.A. et al. In-hospital stroke in patients after cardiac surgery or invasive interventions // Nevrologiya, neiropsikhiatriya, psikhosomatika (Neurology, Neuropsychiatry, Psychosomatics). – 2017. – V. 9. – No. 4. – P. 38–45. In Russian]. DOI: 0.14412/2074-2711-2017-4-38-45.
18. Астраков С.В. и др. Нейрореанимационный этап лечения ишемических инсультов. – Новосибирск: Новосибирский государственный университет, 2015. – С. 60. [Astrakov S.V. et al. Neuroreanimation stage in the treatment of ischemic strokes. Novosibirsk: Novosibirsk State University, 2015. – P. 60. In Russian].
19. Ершов В.И. и др. Математическое моделирование ишемического инсульта // Неврология, нейропсихиатрия, психосоматика. – 2019. – Т. 11. – № 4. – С. 38–43. [Ershov V.I. et al. Mathematic modeling of ischemic stroke // Nevrologiya, neiropsikhiatriya, psikhosomatika (Neurology, Neuropsychiatry, Psychosomatics). – 2019. – V. 11. – No. 4. – P. 38–43. In Russian]. DOI: 10.14412/2074-2711-2019-4-38-43.
20. Дац А.В. и др. Влияние гиперволемии на тяжесть состояния, органные повреждения и выживаемость пациентов отделений интенсивной терапии // Политравма. – 2012. – № 2. – С. 31–34. [Dats A.V. et al. Influence of hypervolemia on severity of state, organ injuries and survival in patients in intensive care unit // Polytrauma. – 2012. – No. 2. – P. 31–34. In Russian].
21. Sanchez M. et al. Comparison of fluid compartments and fluid responsiveness in septic and non-septic patients // Anaesth. Intensive Care. – 2011. –V. 39. – No. 6. DOI: 10.1177/0310057X1103900607.
22. Кунцевич Г.И. и др. Клиническое значение комплексного ультразвукового исследования сердечно-сосудистой системы в остром периоде ишемического инсульта // Ультразвуковая диагностика. – 2000. – № 4. – С. 42–48. [Kuntcevich G.I. et al. Clinical significance of cardiovascular doppler ultrasonography in acute period of ischemic stroke // Ultrazvukovaya Diagnostika (Ultrasound Diagnostic). – 2000. – No. 4. – P. 42–48. In Russian].
23. Мареев В.Ю. и др. Клинические рекомендации ОССН – РКО – РНМОТ. Сердечная недостаточность: хроническая (ХСН) и острая декомпенсированная (ОДСН). Диагностика, профилактика и лечение // Кардиология. – 2018. – № 58 (6S). – С. 8–158. [Mareev V.Yu. et al. Russian Heart Failure Society, Russian Society of Cardiology. Russian Scientific Medical Society of Internal Medicine Guidelines for Heart failure: chronic (CHF) and acute decompensated (ADHF). Diagnosis, prevention and treatment // Kardiologiia (Cardiology). – 2018. – No. 58 (6S). – P. 8–158. In Russian]. DOI: 10.18087/cardio.2475.
24. Чечулин А.Д. Оценка волемического статуса пациентов с острыми цереброваскулярными болезнями // Актуальные вопросы современной медицины. Материалы VI Международной научно-практической конференции прикаспийских государств. – Астрахань: АГМУ, 2022. – С. 101–108. [Chechulin A.D. Evaluation of the volemic status of patients with acute vascular diseases // Current issues of modern medicine. Materials of the VI International Scientific and Practical Conference of the Caspian Littoral States. – Astrakhan: ASMU, 2022. – P. 101–108. In Russian].
25. Latta T. Malignant cholera: documents, communicated by the central board of health, London, relative to the treatment of cholera by the copious injection of aqueous and saline fluid into the veins // Lancet. – 1832. – V. 18. – No. 457. DOI: 10.1016/S0140-6736(02)80289-6.
26. Хейнц У. и др. Водно-электролитный и кислотно-основной баланс. – Москва: Бином, 2015. – С. 359. [Heitz U. et al. Fluid, electrolyte, and acid-base balance. – Moscow: Binom, 2015. – P. 359. In Russian].
27. Пеплоу Ф.В. и др. Биомаркеры инсульта. – Москва: ГЭОТАР-Медиа, 2022. – С. 508. [Peplow P.V. et al. Stroke Biomarkers. – Moscow: GEOTAR-Media, 2022. – P. 508. In Russian].
28. Newaz M. et al. Natriuretic and renoprotective effect of chronic oral neutral endopeptidase inhibition in acute renal failure // Renal failure. – 2010. – V. 32. – No. 3. – P. 384–390.
29. Makikallio A.M. et al. Natriuretic peptides and mortality after stroke // Stroke. – 2005. – V. 36. – P. 1016–1020.
30. Karthikeyan G. et al. A pre-operative brain natriuretic peptide or N-terminal pro-B-type natriuretic peptide measurement an independent predictor of adverse cardiovascular outcomes within 30 days of noncardiac surgery? A systematic review and meta-analysis of observational studies // J. Am. Coll. Cardiol. – 2009. – V. 54. – No. 17. – P. 1599–1606. DOI: 10.1016/j.jacc.2009.06.028.
31. Pandit K. et al. Natriuretic peptides: Diagnostic and therapeutic use // Indian J. Endocrinol. Metab. – 2011. – V. 15. – Suppl. 4. – P. 345–353. DOI: 10.4103/2230-8210.86978.
32. Shibazaki K. et al. Differences in brain natriuretic peptide value between transient ischemic attack and stroke patients with atrial fibrillation // Eur. Neurol. – 2011. – V. 66. – P. 271–276. DOI: 10.1159/000331483.
33. Shibazaki K. et al. Plasma brain natriuretic peptide can be a biological marker to distinguish cardioembolic stroke from other stroke types in acute ischemic stroke // Intern. Med. – 2009. – V. 48. – No. 5. – P. 259–264. DOI: 10.2169/internalmedicine.48.1475.
34. Shimizu H. et al. High plasma brain natriuretic polypeptide level as a marker of risk for thromboembolism in patients with nonvalvular atrial fibrillation // Stroke. – 2002. – V. 33. – P. 1005–1010. DOI: 10.1161/hs0402.105657.
35. Whiteley W. et al. Blood markers and poor outcome after acute cerebrovascular disease: a prospective cohort study // J. Neurol. Neurosurg. Psychiatry. – 2010. – V. 81. – No. 11. – P. 70–85.
36. Montaner J. et al. A panel of biomarkers including caspase-3 and D-dimer may differentiate acute stroke from stroke-mimicking conditions in the emergency department // J. Intern. Med. – 2011. – V. 270. – No. 2. – P. 166–174. DOI: 10.1111/j.1365-2796.2010.02329.x.
37. Casco V.H. et al. Natriuretic peptide system gene expression in human coronary arteries // J. Histochem. Cytochem. – 2002. – V. 50. – No. 6. – P. 799–809. DOI: 10.1177/002215540205000606.
38. Naya T. et al. Brain natriuretic peptide as a surrogate marker for cardioembolic stroke with paroxysmal atrial fibrillation // Cerebrovasc. Dis. – 2008. – V. 26. – No. 4. – P. 434–440. DOI: 10.1159/000155640.
39. Mohammed G.E. et al. The conundrum of volume status assessment: revisiting current and future tools available for physicians at the bedside // Cureus. – 2021. – V. 13. – No. 5. DOI: 10.7759/cureus.15253.
40. Wang C.S. Does this dyspneic patient in the emergency department have congestive heart failure? // JAMA. – 2005. DOI: 10.1001/jama.294.15.1944.
41. Аксельрод Б.А. и др. Влияние волемического статуса на концентрацию предсердного натрийуретического пептида у кардиохирургических пациентов // Кардиология и сердечно-сосудистая хирургия. – 2023. – № 1. – Т. 16. – С. 32–38. [Akselrod B.A. et al. Influence of volemic status on serum atrial natriuretic peptide in cardiac surgical patients // Kard. i serd.-sosud. khir. (Russ. Jour. of Card. and Cardiovasc. Surg). – 2023. – No. 1. – V.16. – P. 32–38. In Russian]. DOI: 10.17116/kardio20231601132.
42. Sivalingam M. et al. The role of natriuretic peptides in volume assessment and mortality prediction in Haemodialysis patients // BMC Nephrol. – 2015. DOI: 10.1186/s12882-015-0212-4.
43. Norman J.A. et al. Degradation of brain natriuretic peptide by neutral endopeptidase: species specific sites of proteolysis determined by mass spectrometry // Biochem. Biophys. Res. Commun. – 1991. DOI: 10.1016/s0006-291x(05)81194-5.
44. Козлов И.А. и др. Повышенный уровень натрийуретического пептида B-типа (NT-proBNP) как фактор риска у кардиохирургических больных // Общая реаниматология. – 2010. – № 1. – С. 49–55. [Kozlov I.A. et al. The higher level of NT-proBNP as a risk factor in cardio-surgical patients // Obsch. Reanimatol. – 2010. – No. 1. – P. 49–55. In Russian].
45. Комаров С.А. и др. Взаимосвязь показателя NT-proBNP с изменениями гемодинамики и выраженностью отека легких при комплексных вмешательствах на клапанах сердца // Вестник анестезиологии и реаниматологии. – 2013. – № 3. – С.18–24. [Komarov S.A. et al. Correlation of NT-proBNP rates with changes in hemodynamics and intensity of pulmonary edema in integrated surgery in cardiac valves // Vestnik Anasteziol. i Reanimatol (Bulletin of Anesthesiology and Reanimatology). – 2013. – No. 3. – P. 18–24. In Russian].
46. Maisel A.S., Daniels L.B. Breathing not properly 10 years later: what we have learned and what we still need to learn // J. Coll. Cardiol. – 2012. – V. 60. – No. 4. – P. 277–282.
47. Bustamante A. et al. Blood biomarkers for the early diagnosis of stroke: the Stroke-Chip Study // Stroke. – 2017. – V. 48. – P. 2419–2425. DOI: 10.1161/strokeaha.117.017076.
48. Ely E.W. et al. Radiologic determination of intravascular volume status using portable, digital chest radiography: a prospective investigation in 100 patients // Crit. Care Med. – 2001. – No. 29. – P. 1502–1512. DOI: 10.1097/00003246-200108000-00002.
49. Saugel B. et al. Physical examination, central venous pressure, and chest radiography for the prediction of transpulmonary thermodilution-derived hemodynamic parameters in critically ill patients: A prospective trial // J. Crit. Care. – 2011. – No. 26. – P. 402–410. DOI: 10.1016/j.jcrc.2010.11.001.
50. Marik P.E. et al. Hemodynamic parameters to guide fluid therapy // Ann. Intensive Care. – 2011. – No. 1. DOI:10.1186/2110-5820-1-1.
51. Eskesen T.G. et al. Systematic review including re-analyses of 1148 individual data sets of central venous pressure as a predictor of fluid responsiveness // Intensive Care Med. – 2016. – No. 42. – P. 324–332. DOI: 10.1007/s00134-015-4168-4.
52. Osman D. et al. Cardiac filling pressures are not appropriate to predict hemodynamic response to volume challenge // Crit. Care Med. – 2007. – No. 35. – P. 64–68. DOI: 10.1097/01.CCM.0000249851.94101.4F.
53. Marik P.E. et al. Does the central venous pressure predict fluid responsiveness? An updated meta-analysis and a plea for some common sense // Crit. Care Med. – 2013. – No. 41. – P. 1774–1781. DOI: 10.1097/CCM.0b013e31828a25fd.
54. Boldt J. et al. Volume replacement strategies on intensive care units: results from a postal survey // Intensive Care Med. – 1998. – No. 24. – P. 147–151. DOI: 10.1007/s001340050536.
55. Kastrup M. et al. Current practice of hemodynamic monitoring and vasopressor and inotropic therapy in post-operative cardiac surgery patients in Germany: results from a postal survey // Acta Anaes. Scand. – 2007. – No. 51. – P. 347–358. DOI: 10.1111/j.1399-6576.2006.01190.x.
56. Marik P.E. et al. Does central venous pressure predict fluid responsiveness? A systematic review of the literature and the tale of seven mares // Chest. – 2008. – No. 134. – P. 172–178. – DOI: 10.1378/chest.07-2331.
57. Marik P.E. et al. Non-invasive hemodynamic monitoring in the intensive care unit // Crit Care Clin. – 2007. – No. 23. – P. 383–400. DOI: 10.1016/j.ccc.2007.05.002.
58. De Backer D. et al. Should we measure the central venous pressure to guide fluid management? Ten answers to 10 questions // Critical Care. – 2018. – No. 22. DOI: 10.1186/s13054-018-1959-3.
59. Дац А.В. и др. Перегрузка жидкостью: причины, диагностика, осложнения, течение // Вестник анестезиологии и реаниматологии. – 2015. – Т. 12. – № 3. – С. 65–73. [Dats A.V. et al. The fluid overload: causes, diagnosis, complications, treatment // Vestnik Anasteziol. i Reanimatol. – 2015. – V. 12. – No. 3. – P. 65–73. In Russian].
60. Самохвалов И.М. и др. Особенности мониторирования гемодинамики у пострадавших с тяжелой сочетанной травмой // Вестник анестезиологии и реаниматологии. – 2015. – Т. 12. – № 3. – С. 34–40. [Samokhvalov I.M. et al. The patterns of the hemodynamics monitoring in the bad concomitant injury patients // Vestnik Anasteziol. i Reanimatol. – 2015. – V. 12. – No. 3. – P. 34–40. In Russian].
61. Kumar A. et al. Pulmonary artery occlusion pressure and central venous pressure fail to predict ventricular filling volume, cardiac performance, or the response to volume infusion in normal subjects // Crit. Care Med. – 2004. – No. 32. – P. 691–699. DOI: 10.1097/01.CCM.0000114996.68110.C9.
62. Irwin R. et al. Techniques and minimally invasive monitoring in intensive care medicine. – New York: Lipincott Williams and Wilkins, 2012. – P. 135.
63. Lichtenstein D.A. et al. A-lines and B-lines: lung ultrasound as a bedside tool for predicting pulmonary artery occlusion pressure in the critically ill // Chest. – 2009. – No. 136. – P. 1014–1020. DOI: 10.1378/chest.09-0001.
64. Dark P.M. et al. The validity of trans-esophageal Doppler ultrasonography as a measure of cardiac output in critically ill adults // Intensive Care Med. – 2004. – No. 30. – P. 2060–2066. – DOI: 10.1007/s00134-004-2430-2.
65. Stec S. et al. First experience with microprobe transoesophageal echocardiography in non-sedated adults undergoing atrial fibrillation ablation: feasibility study and comparison with intracardiac echocardiography // Europace. – 2011. – No. 13. – P. 51–56. DOI: 10.1093/europace/euq349.
66. Ильина Я.Ю. и др. Прогнозирование ответа на инфузионную нагрузку: современные подходы и перспективы // Вестник анестезиологии и реаниматологии. – 2017. – Т. 14. – No. 3. – С. 25–34. [Ilyina Ya.Yu. et al. Predicting response to fluid administration: current approaches and trends // Messenger of Anesthesiology and Resuscitation. – 2017. – V. 14. – No. 3. – P. 25–34. In Russian]. DOI: 10.21292/2078-5658-2017-14-3-25-34.
67. Lyon M. et al. Sonographic measurement of the inferior vena cava as a marker of blood loss // Am. J. Emerg. Med. – 2005. – No. 23. – P. 45–50. DOI: 10.1016/j.ajem.2004.01.004.
68. Cheriex E.C. et al. Echography of the inferior vena cava is a simple and reliable tool for estimation of ‘dry weight’ in haemodialysis patients // Nephrol. Dial. Transplant. – 1989. – No. 4. – P. 563–568.
69. Koratala A. et al. Point-of-care ultrasonography for objective volume management in end-stage renal disease // Blood Purif. – 2020. – No. 49. – P. 132–136. DOI: 10.1159/000503000.
70. Ponikowski P. et al. 2016 ESC Guidelines for the diagnosis and treatment of acute and chronic heart failure: the Task Force for the diagnosis and treatment of acute and chronic heart failure of the European Society of Cardiology (ESC). Developed with the special contribution of the Heart Failure Association (HFA) of the ESC // Eur. J. Heart Fail. – 2016. – No. 18. – P. 891–975. DOI: 10.1002/ejhf.592.
71. Асланова Р.Ш. и др. Клиническая роль ультразвуковой оценки венозного печеночного застоя (VExUS) у пациента с декомпенсацией хронической сердечной недостаточности и сердечно-печеночным синдромом // Клиническая фармакология и терапия. – 2021. № 30. – С. 76–80. [Aslanova R.Sh. et al. Ultrasound evaluation of venous hepatic congestion assessed by VExUS in a patient with decompensated chronic heart failure and cardio-hepatic syndrome // Klinicheskaya farmakologiya (Clin Pharmacol Ther). – 2021. – No. 30. – P. 76–80. In Russian]. DOI: 10.32756/0869-5490-2021-3-76-80.
72. Beaubien-Souligny W. et al. Quantifying systemic congestion with Point-Of-Care ultrasound: development of the venous excess ultrasound grading system // Ultrasound J. – 2020. – No. 12. – DOI: 10.1186/s13089-020-00163-w.
73. Махарин О.А. и др. Фокусированная эхокардиография в мониторинге угрожающих жизни состояний: учебное пособие. – Ростов-на-Дону: Изд-во РостГМУ, 2021. – С. 99. [Makharin O.A. et al. Focused echocardiography in monitoring life-threatening conditions: Tutorial // Rostov-on-Don: Publishing company of RostSMU, 2021. – P. 99. In Russian].
74. Киллу К. и др. УЗИ в отделении интенсивной терапии. – Москва: ГЭОТАР-Медиа, 2016. – С. 143. [Killu K. et al. The ICU Ultrasound. – Moscow: GEOTAR-Media, 2016. – P. 143. In Russian].
75. Старостин Д.О. и др. Роль ультразвука в оценке волемического статуса пациентов в критических состояниях // Вестник интенсивной терапии имени А.И. Салтанова. – 2018. – № 4. – C. 42–50. [Starostin D.O. et al. Role of ultrasound in diagnosing volume status in critically ill patients // Alexander Saltanov Intensive Care Herald. – 2018. – No. 4. – P. 42–50. In Russian].
76. Нигматуллина А. и др. Влияние изменения положения тела человека на величину диаметра внутренних яремных вен // Фундаментальные исследования. – 2015. – № 1. – С. 120–123. [Nigmatullina A. et al. The effect of changing the position of the human body on the diameter of the internal jugular veins // Fundamental research. – 2015. – No. 1. – P. 120–123. In Russian].
77. McLean A.S. Echocardiography in shock management // Crit. Care. – 2016. DOI: 10.1186/s13054-016-1401-7.
78. Lichtenstein D.A. et al. Relevance of lung ultrasound in the diagnosis of acute respiratory failure: the BLUE protocol // Chest. – 2008. – No. 134. – P. 117–125. DOI: 10.1378/chest.07-2800.
79. Picano E. et al. Ultrasound lung comets: a clinically useful sign of extravascular lung water // J. Am. Soc. Echocardiogr. – 2006. – No. 19. – P. 356–363. DOI: 10.1016/j.echo.2005.05.019.
80. Jambrik Z. et al. Usefulness of ultrasound lung comets as a nonradiologic sign of extravascular lung water // Am. J. Cardiol. – 2004. – No. 93. – P. 1265–1270. DOI:10.1016/j.amjcard.2004.02.012.
81. Soldati G. et al. The use of lung ultrasound images for the differential diagnosis of pulmonary and cardiac interstitial pathology // J. Ultrasound. – 2017. – No. 20. – P. 91–96. DOI: 10.1007/s40477-017-0244-7.
82. Kameda T. et al. Point-of-care lung ultrasound for the assessment of pneumonia: a narrative review in the COVID-19 era // J. Med. Ultrason. – 2021. – No. 48. – P. 31–43. DOI: 10.1007/s10396-020-01074-y.
83. Mohammad W.H. et al. Chest ultrasound as a new tool for assessment of volume status in hemodialysis patients // Saudi J. Kidney Dis. Transpl. – 2020. – No. 31. – P. 805–813. DOI: 10.4103/1319-2442.292314.
84. Chiara Busti et al. Ultrasound in the diagnosis of stroke-associated pneumonia // Internal and Emergency Medicine. – 2012. – No. 9. – P. 173–178. DOI: 10.1007/s11739-012-0832-7.
85. Jaffrin M.Y. et al. Body fluid volumes measurements by impedance: a review of bioimpedance spectroscopy (BIS) and bioimpedance analysis (BIA) methods // Med. Eng. Phys. – 2008. – No. 30. – P. 1257–1269. DOI: 10.1016/j.medengphy.2008.06.009.
86. Amir O. et al. A novel approach to monitoring pulmonary congestion in heart failure: initial animal and clinical experiences using remote dielectric sensing technology // Congest Heart Fail. – 2013. – No. 19. – P. 149–155.
87. Amir O. et al. Validation of remote dielectric sensing (ReDStm) technology for quantification of lung fluid status: Comparison to high resolution chest computed tomography in patients with and without acute heart failure // Int. J. Cardiol. – 2016. – No. 221. – P. 841–846. DOI: 10.1016/j.ijcard.2016.06.323.
88. Amir O. et al. Evaluation of remote dielectric sensing (ReDS) technology-guided therapy for decreasing heart failure re-hospitalizations // Int. J. Cardiol. – 2017. – No. 240. – P. 279–284. DOI: 10.1016/j.ijcard.2017.02.120.
89. Uriel N. et al. Relationship between noninvasive assessment of lung fluid volume and invasively measured cardiac hemodynamics // J. Am. Heart Assoc. – 2018. – No. 7. DOI: 10.1161/JAHA.118.009175.
90. Abraham W.T. et al. Primary results of the sensible medical innovations lung fluid status monitor allows reducing readmission rate of heart failure patients (SMILE) trial // J. Card. Fail. – 2019. – No. 25. DOI: 10.1016/j.cardfail.2019.11.007.
91. Bensimhon D. et al. The use of the reds noninvasive lung fluid monitoring system to assess readiness for discharge in patients hospitalized with acute heart failure: a pilot study // Heart and Lung. – 2021. – No. 50. – P. 59–64. DOI: 10.1016/j.hrtlng.2020.07.003.
92. Lala A. et al. Early use of remote dielectric sensing after hospitalization to reduce heart failure readmissions // ESC Heart Fail. – 2021. – No. 8. – P. 1047–1054. DOI: 10.1002/ehf2.13026.
93. Лясникова Е.А. и др. Менеджмент больных с хронической сердечной недостаточностью в Российской Федерации: горизонты и реалии второй декады XXI века // Российский кардиологический журнал. – 2021. – № 26. [Lyasnikova E.A. et al. Management of heart failure patients in Russia: perspectives and realities of the second decade of the XXI century // Russian Journal of Cardiology. – 2021. – No. 26. In Russian]. DOI:10.15829/1560- 4071-2021-4658.
94. Chih-Jun Lai et al. Detecting volemic, cardiac, and autonomic responses from hypervolemia to normovolemia via non-invasive ClearSight hemodynamic monitoring during hemodialysis: an observational investigation // Front. Physiol. – 2022. – V. 13. DOI:10.3389/fphys.2022.775631.
95. Muller L. et al. An increase in aortic blood flow after an infusion of 100 ml colloid over 1 min can predict fluid responsiveness // Anesthesiology. – 2011. – V. 115. – P. 541–547. DOI: 10.1097/ALN.0b013e318229a500.
96. Teboul J.L. et al. Prediction of volume responsiveness in critically ill patients with spontaneous breathing activity // Curr. Opin. Crit. Care. – 2008. – No. 14. – P. 334–349. DOI: 10.1097/MCC.0b013e3282fd6e1e.
97. Monnet X. et al. Passive leg raising for predicting fluid responsiveness: importance of the postural change // Intens. Care Med. – 2009. – V. 35. – P. 85–90. DOI:10.1007/s00134-008-1293-3.
2. Cordemans C. et al. Aiming for a negative fluid balance in patients with acute lung injury and increased intra-abdominal pressure: a pilot study looking at the effects of PAL-treatment // Ann. Intensive Care. – 2012. – No. 5. – Suppl. 1. DOI: 10.1186/2110-5820-2-S1-S15.
3. Бобовник С.В. и др. Периоперационная инфузионная терапия у взрослых // Анестезиология и реаниматология. – 2021. – № 4. – С. 17–33. [Bobovnik S.V. et al. Perioperative fluid therapy in adults // Anesteziologiya I Reanimatologiya (Russian Journal of Anaesthesiology and Reanimatology). – 2021. – No. 4. – P. 17–33. In Russain]. DOI: 10.17116/anaesthesiology202104117.
4. Методические рекомендации Министерства здравоохранения РФ. Принципы периоперационной инфузионной терапии у взрослых пациентов. – 2018. – С. 57. [Methodological recommendations of the Ministry of Health of the Russian Federation. Principles of perioperative infusion therapy in adult patients. – 2018. – P. 57. In Russian].
5. Максимович Н.Е. и др. Патофизиология системы крови и гемостаза. – Гродно: УО «ГрГМУ», 2010. – С. 344. [Maksimovich N.E. et al. Pathophysiology of the blood system and hemostasis. Grodno: EI «GSMU», 2010. – P. 344. In Russian].
6. Rosenberg A.L. et al. Review of a large clinical series: association of cumulative fluid balance on outcome in acute lung injury: a retrospective review of the ARDSnet tidal volume study cohort // J. Intensive Care Med. – 2009. – V. 24. – No. 1. – P. 35–46. DOI: 10.1177/0885066608329850.
7. Кузьков В.В. и др. Инвазивный мониторинг гемодинаники. – Архангельск: Северный государственный медицинский университет, 2008. – С. 392. [Kuzkov V.V. et al. Invasive Hemodynamic Monitoring. Arhangelsk: Northern State Medical University, 2008. – P. 392. In Russian].
8. Задворнов А.А. и др. Клиническая патофизиология отека головного мозга (часть 1) // Вестник анестезиологии и реаниматологии. – 2017. – Т. 14. – № 3. – С. 44–50. [Zadvornov A.A. et al. Clinical pathophysiology of celebral edems (part 1) // Messenger of Anesthesiology and Resuscitation. – 2017. – V. 14. – No. 3. – P. 44–50. In Russian]. DOI: 10.21292/2078-5658-2017-14-3-44-50.
9. Ершов В.И. и др. Гиповолемическая гипернатриемия в дебюте тяжелого ишемического инсульта как предиктор неблагоприятного исхода // Клиника нервных и психических заболеваний. – 2016. – № 6. – С. 10–13. [Ershov V.I. et al. Hypovolemic hyponatremia at the onset of severe ischemic stroke as a predictor of adverse outcome // Zhurnal Nevrologii i Psikhiatrii imeni S.S. Korsakova. – 2016. – No. 6. – P. 10–13. In Russian]. DOI: 10.17116/jnevro20161166110-13.
10. Widimsky P. What is new in ischemic stroke? // Dialogues in Cardiovascular Medicine. – 2017. – V. 22. – No. 3. – P. 25–26.
11. Гончар И.А. Биохимические предикторы и маркеры инфаркта головного мозга. – Минск: БелМАПО, 2013. – С. 512. [Gontschar I.A. et al. Biochemical predictors and markers of ischemic stroke. – Minsk: BMAPE, 2013. – P. 512. In Russian].
12. Nag S. et al. Review: molecular pathogenesis of blood-brain barrier breakdown in acute brain injury // Neuropathology and Applied Neurobiology. – 2011. – V. 37. – No. 9. – P. 3–23.
13. Stokum J.A. et al. Molecular pathophysiology of cerebral edema // Journal Blood Flow Metabolism. – 2016. – V. 36. – No. 3. – P. 513–538.
14. Stokum J.A. et al. Mechanisms of astrocyte-mediated cerebral edema // Neurochemical Research. – 2015. – V. 40. – No. 2. – P. 317–328.
15. Thrane A.S. et al. Drowning stars: reassessing the role of astrocytes in brain edema // Trends in Neuroscience. – 2014. – V. 37. – No. 11. – P. 620–628.
16. Кулеш А.А. и др. Связь между интегральной оценкой магнитно-резонансных маркеров церебральной болезни мелких сосудов, клиническим и функциональным статусом в остром периоде ишемического инсульта // Неврология, нейропсихиатрия, психосоматика. – 2018. – Т. 10. – № 1. – С. 24–31. [Kulesh A.A. et al. A relationship between the integrated assessment of magnetic resonance imaging markers for cerebral small vessel disease and the clinical and functional status in the acute period of ischemic stroke // Nevrologiya, neiropsikhiatriya, psikhosomatika (Neurology, Neuropsychiatry, Psychosomatics). – 2018. – V. 10. – No. 1. – P. 24–31. In Russian]. DOI: 0.14412/2074-2711-2018-1-24-31.
17. Филимонова П.А. и др. Внутрибольничный инсульт у пациентов после кардиохирургических операций и инвазивных вмешательств на сердце // Неврология, нейропсихиатрия, психосоматика. – 2017. – Т. 9. – № 4. – С. 38–45. [Filimonova P.A. et al. In-hospital stroke in patients after cardiac surgery or invasive interventions // Nevrologiya, neiropsikhiatriya, psikhosomatika (Neurology, Neuropsychiatry, Psychosomatics). – 2017. – V. 9. – No. 4. – P. 38–45. In Russian]. DOI: 0.14412/2074-2711-2017-4-38-45.
18. Астраков С.В. и др. Нейрореанимационный этап лечения ишемических инсультов. – Новосибирск: Новосибирский государственный университет, 2015. – С. 60. [Astrakov S.V. et al. Neuroreanimation stage in the treatment of ischemic strokes. Novosibirsk: Novosibirsk State University, 2015. – P. 60. In Russian].
19. Ершов В.И. и др. Математическое моделирование ишемического инсульта // Неврология, нейропсихиатрия, психосоматика. – 2019. – Т. 11. – № 4. – С. 38–43. [Ershov V.I. et al. Mathematic modeling of ischemic stroke // Nevrologiya, neiropsikhiatriya, psikhosomatika (Neurology, Neuropsychiatry, Psychosomatics). – 2019. – V. 11. – No. 4. – P. 38–43. In Russian]. DOI: 10.14412/2074-2711-2019-4-38-43.
20. Дац А.В. и др. Влияние гиперволемии на тяжесть состояния, органные повреждения и выживаемость пациентов отделений интенсивной терапии // Политравма. – 2012. – № 2. – С. 31–34. [Dats A.V. et al. Influence of hypervolemia on severity of state, organ injuries and survival in patients in intensive care unit // Polytrauma. – 2012. – No. 2. – P. 31–34. In Russian].
21. Sanchez M. et al. Comparison of fluid compartments and fluid responsiveness in septic and non-septic patients // Anaesth. Intensive Care. – 2011. –V. 39. – No. 6. DOI: 10.1177/0310057X1103900607.
22. Кунцевич Г.И. и др. Клиническое значение комплексного ультразвукового исследования сердечно-сосудистой системы в остром периоде ишемического инсульта // Ультразвуковая диагностика. – 2000. – № 4. – С. 42–48. [Kuntcevich G.I. et al. Clinical significance of cardiovascular doppler ultrasonography in acute period of ischemic stroke // Ultrazvukovaya Diagnostika (Ultrasound Diagnostic). – 2000. – No. 4. – P. 42–48. In Russian].
23. Мареев В.Ю. и др. Клинические рекомендации ОССН – РКО – РНМОТ. Сердечная недостаточность: хроническая (ХСН) и острая декомпенсированная (ОДСН). Диагностика, профилактика и лечение // Кардиология. – 2018. – № 58 (6S). – С. 8–158. [Mareev V.Yu. et al. Russian Heart Failure Society, Russian Society of Cardiology. Russian Scientific Medical Society of Internal Medicine Guidelines for Heart failure: chronic (CHF) and acute decompensated (ADHF). Diagnosis, prevention and treatment // Kardiologiia (Cardiology). – 2018. – No. 58 (6S). – P. 8–158. In Russian]. DOI: 10.18087/cardio.2475.
24. Чечулин А.Д. Оценка волемического статуса пациентов с острыми цереброваскулярными болезнями // Актуальные вопросы современной медицины. Материалы VI Международной научно-практической конференции прикаспийских государств. – Астрахань: АГМУ, 2022. – С. 101–108. [Chechulin A.D. Evaluation of the volemic status of patients with acute vascular diseases // Current issues of modern medicine. Materials of the VI International Scientific and Practical Conference of the Caspian Littoral States. – Astrakhan: ASMU, 2022. – P. 101–108. In Russian].
25. Latta T. Malignant cholera: documents, communicated by the central board of health, London, relative to the treatment of cholera by the copious injection of aqueous and saline fluid into the veins // Lancet. – 1832. – V. 18. – No. 457. DOI: 10.1016/S0140-6736(02)80289-6.
26. Хейнц У. и др. Водно-электролитный и кислотно-основной баланс. – Москва: Бином, 2015. – С. 359. [Heitz U. et al. Fluid, electrolyte, and acid-base balance. – Moscow: Binom, 2015. – P. 359. In Russian].
27. Пеплоу Ф.В. и др. Биомаркеры инсульта. – Москва: ГЭОТАР-Медиа, 2022. – С. 508. [Peplow P.V. et al. Stroke Biomarkers. – Moscow: GEOTAR-Media, 2022. – P. 508. In Russian].
28. Newaz M. et al. Natriuretic and renoprotective effect of chronic oral neutral endopeptidase inhibition in acute renal failure // Renal failure. – 2010. – V. 32. – No. 3. – P. 384–390.
29. Makikallio A.M. et al. Natriuretic peptides and mortality after stroke // Stroke. – 2005. – V. 36. – P. 1016–1020.
30. Karthikeyan G. et al. A pre-operative brain natriuretic peptide or N-terminal pro-B-type natriuretic peptide measurement an independent predictor of adverse cardiovascular outcomes within 30 days of noncardiac surgery? A systematic review and meta-analysis of observational studies // J. Am. Coll. Cardiol. – 2009. – V. 54. – No. 17. – P. 1599–1606. DOI: 10.1016/j.jacc.2009.06.028.
31. Pandit K. et al. Natriuretic peptides: Diagnostic and therapeutic use // Indian J. Endocrinol. Metab. – 2011. – V. 15. – Suppl. 4. – P. 345–353. DOI: 10.4103/2230-8210.86978.
32. Shibazaki K. et al. Differences in brain natriuretic peptide value between transient ischemic attack and stroke patients with atrial fibrillation // Eur. Neurol. – 2011. – V. 66. – P. 271–276. DOI: 10.1159/000331483.
33. Shibazaki K. et al. Plasma brain natriuretic peptide can be a biological marker to distinguish cardioembolic stroke from other stroke types in acute ischemic stroke // Intern. Med. – 2009. – V. 48. – No. 5. – P. 259–264. DOI: 10.2169/internalmedicine.48.1475.
34. Shimizu H. et al. High plasma brain natriuretic polypeptide level as a marker of risk for thromboembolism in patients with nonvalvular atrial fibrillation // Stroke. – 2002. – V. 33. – P. 1005–1010. DOI: 10.1161/hs0402.105657.
35. Whiteley W. et al. Blood markers and poor outcome after acute cerebrovascular disease: a prospective cohort study // J. Neurol. Neurosurg. Psychiatry. – 2010. – V. 81. – No. 11. – P. 70–85.
36. Montaner J. et al. A panel of biomarkers including caspase-3 and D-dimer may differentiate acute stroke from stroke-mimicking conditions in the emergency department // J. Intern. Med. – 2011. – V. 270. – No. 2. – P. 166–174. DOI: 10.1111/j.1365-2796.2010.02329.x.
37. Casco V.H. et al. Natriuretic peptide system gene expression in human coronary arteries // J. Histochem. Cytochem. – 2002. – V. 50. – No. 6. – P. 799–809. DOI: 10.1177/002215540205000606.
38. Naya T. et al. Brain natriuretic peptide as a surrogate marker for cardioembolic stroke with paroxysmal atrial fibrillation // Cerebrovasc. Dis. – 2008. – V. 26. – No. 4. – P. 434–440. DOI: 10.1159/000155640.
39. Mohammed G.E. et al. The conundrum of volume status assessment: revisiting current and future tools available for physicians at the bedside // Cureus. – 2021. – V. 13. – No. 5. DOI: 10.7759/cureus.15253.
40. Wang C.S. Does this dyspneic patient in the emergency department have congestive heart failure? // JAMA. – 2005. DOI: 10.1001/jama.294.15.1944.
41. Аксельрод Б.А. и др. Влияние волемического статуса на концентрацию предсердного натрийуретического пептида у кардиохирургических пациентов // Кардиология и сердечно-сосудистая хирургия. – 2023. – № 1. – Т. 16. – С. 32–38. [Akselrod B.A. et al. Influence of volemic status on serum atrial natriuretic peptide in cardiac surgical patients // Kard. i serd.-sosud. khir. (Russ. Jour. of Card. and Cardiovasc. Surg). – 2023. – No. 1. – V.16. – P. 32–38. In Russian]. DOI: 10.17116/kardio20231601132.
42. Sivalingam M. et al. The role of natriuretic peptides in volume assessment and mortality prediction in Haemodialysis patients // BMC Nephrol. – 2015. DOI: 10.1186/s12882-015-0212-4.
43. Norman J.A. et al. Degradation of brain natriuretic peptide by neutral endopeptidase: species specific sites of proteolysis determined by mass spectrometry // Biochem. Biophys. Res. Commun. – 1991. DOI: 10.1016/s0006-291x(05)81194-5.
44. Козлов И.А. и др. Повышенный уровень натрийуретического пептида B-типа (NT-proBNP) как фактор риска у кардиохирургических больных // Общая реаниматология. – 2010. – № 1. – С. 49–55. [Kozlov I.A. et al. The higher level of NT-proBNP as a risk factor in cardio-surgical patients // Obsch. Reanimatol. – 2010. – No. 1. – P. 49–55. In Russian].
45. Комаров С.А. и др. Взаимосвязь показателя NT-proBNP с изменениями гемодинамики и выраженностью отека легких при комплексных вмешательствах на клапанах сердца // Вестник анестезиологии и реаниматологии. – 2013. – № 3. – С.18–24. [Komarov S.A. et al. Correlation of NT-proBNP rates with changes in hemodynamics and intensity of pulmonary edema in integrated surgery in cardiac valves // Vestnik Anasteziol. i Reanimatol (Bulletin of Anesthesiology and Reanimatology). – 2013. – No. 3. – P. 18–24. In Russian].
46. Maisel A.S., Daniels L.B. Breathing not properly 10 years later: what we have learned and what we still need to learn // J. Coll. Cardiol. – 2012. – V. 60. – No. 4. – P. 277–282.
47. Bustamante A. et al. Blood biomarkers for the early diagnosis of stroke: the Stroke-Chip Study // Stroke. – 2017. – V. 48. – P. 2419–2425. DOI: 10.1161/strokeaha.117.017076.
48. Ely E.W. et al. Radiologic determination of intravascular volume status using portable, digital chest radiography: a prospective investigation in 100 patients // Crit. Care Med. – 2001. – No. 29. – P. 1502–1512. DOI: 10.1097/00003246-200108000-00002.
49. Saugel B. et al. Physical examination, central venous pressure, and chest radiography for the prediction of transpulmonary thermodilution-derived hemodynamic parameters in critically ill patients: A prospective trial // J. Crit. Care. – 2011. – No. 26. – P. 402–410. DOI: 10.1016/j.jcrc.2010.11.001.
50. Marik P.E. et al. Hemodynamic parameters to guide fluid therapy // Ann. Intensive Care. – 2011. – No. 1. DOI:10.1186/2110-5820-1-1.
51. Eskesen T.G. et al. Systematic review including re-analyses of 1148 individual data sets of central venous pressure as a predictor of fluid responsiveness // Intensive Care Med. – 2016. – No. 42. – P. 324–332. DOI: 10.1007/s00134-015-4168-4.
52. Osman D. et al. Cardiac filling pressures are not appropriate to predict hemodynamic response to volume challenge // Crit. Care Med. – 2007. – No. 35. – P. 64–68. DOI: 10.1097/01.CCM.0000249851.94101.4F.
53. Marik P.E. et al. Does the central venous pressure predict fluid responsiveness? An updated meta-analysis and a plea for some common sense // Crit. Care Med. – 2013. – No. 41. – P. 1774–1781. DOI: 10.1097/CCM.0b013e31828a25fd.
54. Boldt J. et al. Volume replacement strategies on intensive care units: results from a postal survey // Intensive Care Med. – 1998. – No. 24. – P. 147–151. DOI: 10.1007/s001340050536.
55. Kastrup M. et al. Current practice of hemodynamic monitoring and vasopressor and inotropic therapy in post-operative cardiac surgery patients in Germany: results from a postal survey // Acta Anaes. Scand. – 2007. – No. 51. – P. 347–358. DOI: 10.1111/j.1399-6576.2006.01190.x.
56. Marik P.E. et al. Does central venous pressure predict fluid responsiveness? A systematic review of the literature and the tale of seven mares // Chest. – 2008. – No. 134. – P. 172–178. – DOI: 10.1378/chest.07-2331.
57. Marik P.E. et al. Non-invasive hemodynamic monitoring in the intensive care unit // Crit Care Clin. – 2007. – No. 23. – P. 383–400. DOI: 10.1016/j.ccc.2007.05.002.
58. De Backer D. et al. Should we measure the central venous pressure to guide fluid management? Ten answers to 10 questions // Critical Care. – 2018. – No. 22. DOI: 10.1186/s13054-018-1959-3.
59. Дац А.В. и др. Перегрузка жидкостью: причины, диагностика, осложнения, течение // Вестник анестезиологии и реаниматологии. – 2015. – Т. 12. – № 3. – С. 65–73. [Dats A.V. et al. The fluid overload: causes, diagnosis, complications, treatment // Vestnik Anasteziol. i Reanimatol. – 2015. – V. 12. – No. 3. – P. 65–73. In Russian].
60. Самохвалов И.М. и др. Особенности мониторирования гемодинамики у пострадавших с тяжелой сочетанной травмой // Вестник анестезиологии и реаниматологии. – 2015. – Т. 12. – № 3. – С. 34–40. [Samokhvalov I.M. et al. The patterns of the hemodynamics monitoring in the bad concomitant injury patients // Vestnik Anasteziol. i Reanimatol. – 2015. – V. 12. – No. 3. – P. 34–40. In Russian].
61. Kumar A. et al. Pulmonary artery occlusion pressure and central venous pressure fail to predict ventricular filling volume, cardiac performance, or the response to volume infusion in normal subjects // Crit. Care Med. – 2004. – No. 32. – P. 691–699. DOI: 10.1097/01.CCM.0000114996.68110.C9.
62. Irwin R. et al. Techniques and minimally invasive monitoring in intensive care medicine. – New York: Lipincott Williams and Wilkins, 2012. – P. 135.
63. Lichtenstein D.A. et al. A-lines and B-lines: lung ultrasound as a bedside tool for predicting pulmonary artery occlusion pressure in the critically ill // Chest. – 2009. – No. 136. – P. 1014–1020. DOI: 10.1378/chest.09-0001.
64. Dark P.M. et al. The validity of trans-esophageal Doppler ultrasonography as a measure of cardiac output in critically ill adults // Intensive Care Med. – 2004. – No. 30. – P. 2060–2066. – DOI: 10.1007/s00134-004-2430-2.
65. Stec S. et al. First experience with microprobe transoesophageal echocardiography in non-sedated adults undergoing atrial fibrillation ablation: feasibility study and comparison with intracardiac echocardiography // Europace. – 2011. – No. 13. – P. 51–56. DOI: 10.1093/europace/euq349.
66. Ильина Я.Ю. и др. Прогнозирование ответа на инфузионную нагрузку: современные подходы и перспективы // Вестник анестезиологии и реаниматологии. – 2017. – Т. 14. – No. 3. – С. 25–34. [Ilyina Ya.Yu. et al. Predicting response to fluid administration: current approaches and trends // Messenger of Anesthesiology and Resuscitation. – 2017. – V. 14. – No. 3. – P. 25–34. In Russian]. DOI: 10.21292/2078-5658-2017-14-3-25-34.
67. Lyon M. et al. Sonographic measurement of the inferior vena cava as a marker of blood loss // Am. J. Emerg. Med. – 2005. – No. 23. – P. 45–50. DOI: 10.1016/j.ajem.2004.01.004.
68. Cheriex E.C. et al. Echography of the inferior vena cava is a simple and reliable tool for estimation of ‘dry weight’ in haemodialysis patients // Nephrol. Dial. Transplant. – 1989. – No. 4. – P. 563–568.
69. Koratala A. et al. Point-of-care ultrasonography for objective volume management in end-stage renal disease // Blood Purif. – 2020. – No. 49. – P. 132–136. DOI: 10.1159/000503000.
70. Ponikowski P. et al. 2016 ESC Guidelines for the diagnosis and treatment of acute and chronic heart failure: the Task Force for the diagnosis and treatment of acute and chronic heart failure of the European Society of Cardiology (ESC). Developed with the special contribution of the Heart Failure Association (HFA) of the ESC // Eur. J. Heart Fail. – 2016. – No. 18. – P. 891–975. DOI: 10.1002/ejhf.592.
71. Асланова Р.Ш. и др. Клиническая роль ультразвуковой оценки венозного печеночного застоя (VExUS) у пациента с декомпенсацией хронической сердечной недостаточности и сердечно-печеночным синдромом // Клиническая фармакология и терапия. – 2021. № 30. – С. 76–80. [Aslanova R.Sh. et al. Ultrasound evaluation of venous hepatic congestion assessed by VExUS in a patient with decompensated chronic heart failure and cardio-hepatic syndrome // Klinicheskaya farmakologiya (Clin Pharmacol Ther). – 2021. – No. 30. – P. 76–80. In Russian]. DOI: 10.32756/0869-5490-2021-3-76-80.
72. Beaubien-Souligny W. et al. Quantifying systemic congestion with Point-Of-Care ultrasound: development of the venous excess ultrasound grading system // Ultrasound J. – 2020. – No. 12. – DOI: 10.1186/s13089-020-00163-w.
73. Махарин О.А. и др. Фокусированная эхокардиография в мониторинге угрожающих жизни состояний: учебное пособие. – Ростов-на-Дону: Изд-во РостГМУ, 2021. – С. 99. [Makharin O.A. et al. Focused echocardiography in monitoring life-threatening conditions: Tutorial // Rostov-on-Don: Publishing company of RostSMU, 2021. – P. 99. In Russian].
74. Киллу К. и др. УЗИ в отделении интенсивной терапии. – Москва: ГЭОТАР-Медиа, 2016. – С. 143. [Killu K. et al. The ICU Ultrasound. – Moscow: GEOTAR-Media, 2016. – P. 143. In Russian].
75. Старостин Д.О. и др. Роль ультразвука в оценке волемического статуса пациентов в критических состояниях // Вестник интенсивной терапии имени А.И. Салтанова. – 2018. – № 4. – C. 42–50. [Starostin D.O. et al. Role of ultrasound in diagnosing volume status in critically ill patients // Alexander Saltanov Intensive Care Herald. – 2018. – No. 4. – P. 42–50. In Russian].
76. Нигматуллина А. и др. Влияние изменения положения тела человека на величину диаметра внутренних яремных вен // Фундаментальные исследования. – 2015. – № 1. – С. 120–123. [Nigmatullina A. et al. The effect of changing the position of the human body on the diameter of the internal jugular veins // Fundamental research. – 2015. – No. 1. – P. 120–123. In Russian].
77. McLean A.S. Echocardiography in shock management // Crit. Care. – 2016. DOI: 10.1186/s13054-016-1401-7.
78. Lichtenstein D.A. et al. Relevance of lung ultrasound in the diagnosis of acute respiratory failure: the BLUE protocol // Chest. – 2008. – No. 134. – P. 117–125. DOI: 10.1378/chest.07-2800.
79. Picano E. et al. Ultrasound lung comets: a clinically useful sign of extravascular lung water // J. Am. Soc. Echocardiogr. – 2006. – No. 19. – P. 356–363. DOI: 10.1016/j.echo.2005.05.019.
80. Jambrik Z. et al. Usefulness of ultrasound lung comets as a nonradiologic sign of extravascular lung water // Am. J. Cardiol. – 2004. – No. 93. – P. 1265–1270. DOI:10.1016/j.amjcard.2004.02.012.
81. Soldati G. et al. The use of lung ultrasound images for the differential diagnosis of pulmonary and cardiac interstitial pathology // J. Ultrasound. – 2017. – No. 20. – P. 91–96. DOI: 10.1007/s40477-017-0244-7.
82. Kameda T. et al. Point-of-care lung ultrasound for the assessment of pneumonia: a narrative review in the COVID-19 era // J. Med. Ultrason. – 2021. – No. 48. – P. 31–43. DOI: 10.1007/s10396-020-01074-y.
83. Mohammad W.H. et al. Chest ultrasound as a new tool for assessment of volume status in hemodialysis patients // Saudi J. Kidney Dis. Transpl. – 2020. – No. 31. – P. 805–813. DOI: 10.4103/1319-2442.292314.
84. Chiara Busti et al. Ultrasound in the diagnosis of stroke-associated pneumonia // Internal and Emergency Medicine. – 2012. – No. 9. – P. 173–178. DOI: 10.1007/s11739-012-0832-7.
85. Jaffrin M.Y. et al. Body fluid volumes measurements by impedance: a review of bioimpedance spectroscopy (BIS) and bioimpedance analysis (BIA) methods // Med. Eng. Phys. – 2008. – No. 30. – P. 1257–1269. DOI: 10.1016/j.medengphy.2008.06.009.
86. Amir O. et al. A novel approach to monitoring pulmonary congestion in heart failure: initial animal and clinical experiences using remote dielectric sensing technology // Congest Heart Fail. – 2013. – No. 19. – P. 149–155.
87. Amir O. et al. Validation of remote dielectric sensing (ReDStm) technology for quantification of lung fluid status: Comparison to high resolution chest computed tomography in patients with and without acute heart failure // Int. J. Cardiol. – 2016. – No. 221. – P. 841–846. DOI: 10.1016/j.ijcard.2016.06.323.
88. Amir O. et al. Evaluation of remote dielectric sensing (ReDS) technology-guided therapy for decreasing heart failure re-hospitalizations // Int. J. Cardiol. – 2017. – No. 240. – P. 279–284. DOI: 10.1016/j.ijcard.2017.02.120.
89. Uriel N. et al. Relationship between noninvasive assessment of lung fluid volume and invasively measured cardiac hemodynamics // J. Am. Heart Assoc. – 2018. – No. 7. DOI: 10.1161/JAHA.118.009175.
90. Abraham W.T. et al. Primary results of the sensible medical innovations lung fluid status monitor allows reducing readmission rate of heart failure patients (SMILE) trial // J. Card. Fail. – 2019. – No. 25. DOI: 10.1016/j.cardfail.2019.11.007.
91. Bensimhon D. et al. The use of the reds noninvasive lung fluid monitoring system to assess readiness for discharge in patients hospitalized with acute heart failure: a pilot study // Heart and Lung. – 2021. – No. 50. – P. 59–64. DOI: 10.1016/j.hrtlng.2020.07.003.
92. Lala A. et al. Early use of remote dielectric sensing after hospitalization to reduce heart failure readmissions // ESC Heart Fail. – 2021. – No. 8. – P. 1047–1054. DOI: 10.1002/ehf2.13026.
93. Лясникова Е.А. и др. Менеджмент больных с хронической сердечной недостаточностью в Российской Федерации: горизонты и реалии второй декады XXI века // Российский кардиологический журнал. – 2021. – № 26. [Lyasnikova E.A. et al. Management of heart failure patients in Russia: perspectives and realities of the second decade of the XXI century // Russian Journal of Cardiology. – 2021. – No. 26. In Russian]. DOI:10.15829/1560- 4071-2021-4658.
94. Chih-Jun Lai et al. Detecting volemic, cardiac, and autonomic responses from hypervolemia to normovolemia via non-invasive ClearSight hemodynamic monitoring during hemodialysis: an observational investigation // Front. Physiol. – 2022. – V. 13. DOI:10.3389/fphys.2022.775631.
95. Muller L. et al. An increase in aortic blood flow after an infusion of 100 ml colloid over 1 min can predict fluid responsiveness // Anesthesiology. – 2011. – V. 115. – P. 541–547. DOI: 10.1097/ALN.0b013e318229a500.
96. Teboul J.L. et al. Prediction of volume responsiveness in critically ill patients with spontaneous breathing activity // Curr. Opin. Crit. Care. – 2008. – No. 14. – P. 334–349. DOI: 10.1097/MCC.0b013e3282fd6e1e.
97. Monnet X. et al. Passive leg raising for predicting fluid responsiveness: importance of the postural change // Intens. Care Med. – 2009. – V. 35. – P. 85–90. DOI:10.1007/s00134-008-1293-3.